potentially lethal damage

Objective To analyze the dose rate effect and potentially lethal damage repair in DNA double strand break repair deficient murine cells ( SCID) irradiated by γ ray.

目的探讨γ射线照射后小鼠DNA双链断裂修复缺陷细胞（SCID）的剂量率效应和潜在致死性损伤的修复。

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In this paper a close relationship among the activity of ADP-ribose transf-erase ( ADPRT), rejoining of DNA single strand breaks ( SSB) and potentially lethal damage repair ( PLDR) in mouse tumor cells has been demonstrated.

本文探讨了ADP&核糖基转移酶（ADPRT）的活性、DNA单链断裂（SSB）重接和细胞潜在致死质损伤修复（PLDR）三者的关系。

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However, wether repairing of damaged telomeres is a mechanism of potentially lethal damage repair has not been clarified until now, and little is understood about the mechanism of telomerase performing repairing at damaged telomeres and modifying apoptosis.

目前对端粒损伤修复是否是潜在致死性损伤修复中重要的机制还未见有学者明确提出，对端粒酶如何参与端粒损伤的修复及凋亡的调控机制了解还很少。

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In this paper it is reported that the recovery from 6-12 Gy X-ray irradiation-induced potentially lethal damage ( PLD) in plateau-phase V_ ( 79) cells, following incubation for 2-6h postirradiation was observed.

本文报道了612Gy X线照射引起坪台期V（79）细胞潜在致死损伤（PLD），经保温26h后可以得到修复。细胞存活率比可提高到1.52.0。

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